News from Far-East ( Finnish)

November 17, 2017

https://www.hs.fi/ulkomaat/art-2000005451797.html

http://news.xinhuanet.com/english/2017-10/25/c_136703369.htm

25.10.2017

KAMPALA, Oct. 24 (Xinhua) — Uganda’s ministry of health on Tuesday said two health workers in the eastern part of the country have developed signs of the deadly Marburg fever.

Jane Aceng, minister of health, told reporters that the health workers interacted with the index case which died on Oct. 11 at Kapchorwa hospital.

“Blood samples have been taken off and sent to Uganda Virus Research Institute for testing,” said Aceng, noting that in the interim, the health workers have been isolated and are being monitored.

The number of cumulative cases currently stands at two – with one confirmed and one probable case, according to the ministry.

The minister said a total of 155 people who got into contact with the index case are being monitored.

The last Marburg outbreak in Uganda was reported in central and western parts of the east African country in 2014.

Marburg is a severe and highly fatal disease caused by a virus from the same family as the one that causes Ebola hemorrhagic fever, according to the World Health Organization.

According to the global health body, the illness caused by Marburg virus begins abruptly, with severe headache and malaise.

 

 

 

 

Even news about an unknown letal disease  5.10. 2017 in Sudan

http://outbreaks.globalincidentmap.com/home.php

http://www.emro.who.int/health-topics/cholera-outbreak/weekly-cholera-updates.html

Ebola 2017

May 21, 2017

http://www.who.int/ebola/en/

WHO: Cholera in Yemen

May 20, 2017

http://www.aljazeera.com/news/2017/05/speed-yemen-cholera-outbreak-unprecedented-170519110837434.html

https://books.google.se/books?id=F233OULwmeMC&pg=PA42&lpg=PA42&dq=H3Cit&source=bl&ots=MYikKIQiAV&sig=J_XNkBOHkaw1_mp7xUYsyvZvEoA&hl=sv&sa=X&ved=0ahUKEwjYyJ2N7f3SAhUCjSwKHaX_BCsQ6AEIWjAM#v=onepage&q=H3Cit&f=false

ZIKA-virus campaigns

March 1, 2017

http://www.thelancet.com/campaigns/zika

WHO: Zika virus report

January 22, 2017

http://www.who.int/emergencies/zika-virus/situation-report/Table1_20012017.PNG?ua=1

 

http://www.who.int/en/

http://emedicine.medscape.com/article/833665-overview

White Phosphorus injure, management

https://www.ncbi.nlm.nih.gov/pubmed/11451604

Burns. 2001 Aug;27(5):492-7.

The management of white phosphorus burns.

Abstract

Phosphorus burns are a rarely encountered chemical burn, typically occurring in battle, industrial accidents, or from fireworks. Death may result even with minimal burn areas. Early recognition of affected areas and adequate resuscitation is crucial. Amongst our 2765 admissions between 1984 and 1998, 326 patients had chemical burns. Seven admissions were the result of phosphorus burns

. Our treatment protocol comprises 1% copper sulfate solution for neutralization and identification of phosphorus particles, copious normal saline irrigation, keeping wounds moist with saline-soaked thick pads even during transportation, prompt debridement of affected areas, porcine skin coverage or skin grafts for acute wound management, as well as intensive monitoring of electrolytes and cardiac function in our burns center. Intravenous calcium gluconate is mandatory for correction of hypocalcemia. Of the seven, one patient died from inhalation injury and the others were scheduled for sequential surgical procedures for functional and cosmetic recovery. Cooling affected areas with tap water or normal saline, prompt removal of phosphorus particles with mechanical debridement, intensive monitoring, and maintenance of electrolyte balance are critical steps in initial management. Fluid resuscitation can be adjusted according to urine output. Early excision and skin autografts summarize our phosphorus burn treatment protocol.

http://www.who.int/nutrition/publications/emergencies/WHO_NHD_99.13/en/

Overview

Severe thiamine deficiency causes, beriberi, which is a disorder typically associated with a diet consisting largely of polished rice. It may also occur when refined wheat flour forms a major part of the diet and among alcoholics and food faddists. Thiamine deficiency can develop within 2-3 months of inadequate consumption; it is characterized either by cardiac involvement with oedema or by peripheral neuropathy, with intermediate forms between these two extremes also occurring. Left untreated, thiamine deficiency leads to disability and death. In the past decade, several refugee populations that were wholly dependent on food aid have developed thiamine deficiency.

This document is intended primarily as a basis for ensuring adequate thiamine intake among populations in emergency settings. It reviews strategies for preventing thiamine deficiency among refugees and analyses factors influencing success and failure. Also included are a review of the literature on the epidemiology of thiamine deficiency and its signs and symptoms; thiamine’s properties and functions and the recommended daily allowance; and a discussion of food sources of this vitamin and its stability.

Thiamine deficiency and its prevention and control in major emergencies is the second in an occasional WHO series on the prevention and control of micronutrient deficiencies during emergencies. Reviews concerning scurvy and pellagra are also available.

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